Combination therapy induces unfolded protein response and cytoskeletal rearrangement leading to mitochondrial apoptosis in prostate cancer

Development of therapeutic resistance is responsible for most prostate cancer (PCa) related mortality. Resistance has been attributed to an acquired or selected cancer stem cell phenotype. Here we report the histone deacetylase inhibitor apicidin (APC) or ER stressor thapsigargin (TG) potentiate pac...

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Publicat a:Mol Oncol
Autors principals: Kumar, Sandeep, Chaudhary, Ajay K., Kumar, Rahul, O'Malley, Jordan, Dubrovska, Anna, Wang, Xinjiang, Yadav, Neelu, Goodrich, David W., Chandra, Dhyan
Format: Artigo
Idioma:Inglês
Publicat: John Wiley and Sons Inc. 2016
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Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4972665/
https://ncbi.nlm.nih.gov/pubmed/27106131
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molonc.2016.03.007
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