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A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain
BACKGROUND: Cav3.2 T-type calcium currents in primary afferents are enhanced in various painful pathological conditions, whereas inhibiting Cav3.2 activity or expression offers a strategy for combating the development of pain hypersensitivity. We have shown that Cav3.2 channel surface density is str...
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| Publicado no: | Mol Pain |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
SAGE Publications
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4955966/ https://ncbi.nlm.nih.gov/pubmed/27130589 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1744806916642444 |
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