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Tighter Control by Chymotrypsin C (CTRC) Explains Lack of Association between Human Anionic Trypsinogen and Hereditary Pancreatitis

The human pancreas expresses two major trypsinogen isoforms, cationic trypsinogen (PRSS1) and anionic trypsinogen (PRSS2). Mutations in PRSS1 cause hereditary pancreatitis by altering cleavage of regulatory nick sites by chymotrypsin C (CTRC) resulting in reduced trypsinogen degradation and increase...

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Pubblicato in:J Biol Chem
Autori principali: Jancsó, Zsanett, Sahin-Tóth, Miklós
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Biochemistry and Molecular Biology 2016
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4933207/
https://ncbi.nlm.nih.gov/pubmed/27129265
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M116.725374
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