A ubiquitylation site in Cockayne syndrome B required for repair of oxidative DNA damage, but not for transcription-coupled nucleotide excision repair

Cockayne syndrome B (CSB), best known for its role in transcription-coupled nucleotide excision repair (TC-NER), contains a ubiquitin-binding domain (UBD), but the functional connection between protein ubiquitylation and this UBD remains unclear. Here, we show that CSB is regulated via site-specific...

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Dettagli Bibliografici
Pubblicato in:Nucleic Acids Res
Autori principali: Ranes, Michael, Boeing, Stefan, Wang, Yuming, Wienholz, Franziska, Menoni, Hervé, Walker, Jane, Encheva, Vesela, Chakravarty, Probir, Mari, Pierre-Olivier, Stewart, Aengus, Giglia-Mari, Giuseppina, Snijders, Ambrosius P., Vermeulen, Wim, Svejstrup, Jesper Q.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2016
Soggetti:
Genome Integrity, Repair and Replication
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4914099/
https://ncbi.nlm.nih.gov/pubmed/27060134
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gkw216
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