Colitis-induced bladder afferent neuronal activation is regulated by BDNF through PLCγ pathway

Lignende værker

• Endogenous BDNF augments NMDA receptor phosphorylation in the spinal cord via PLCγ, PKC, and PI3K/Akt pathways during colitis
  af: Liu, Miao, et al.
  Udgivet: (2015)
• Phospholipase C gamma mediates endogenous brain-derived neurotrophic factor-regulated calcitonin gene-related peptide expression in colitis-induced visceral pain
  af: Hashmi, Fiza, et al.
  Udgivet: (2016)
• Suppression of the PI3K pathway in vivo reduces cystitis-induced bladder hypertrophy and restores bladder capacity examined by magnetic resonance imaging.
  af: Zhongwei Qiao, et al.
  Udgivet: (2014-01-01)
• Suppression of the PI3K Pathway In Vivo Reduces Cystitis-Induced Bladder Hypertrophy and Restores Bladder Capacity Examined by Magnetic Resonance Imaging
  af: Qiao, Zhongwei, et al.
  Udgivet: (2014)
• Up-regulation of brain-derived neurotrophic factor is regulated by extracellular signal-regulated protein kinase 5 and by nerve growth factor retrograde signaling in colonic afferent neurons in colitis
  af: Yu, Sharon J, et al.
  Udgivet: (2012)