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A faulty interaction between SOD1 and hCCS in neurodegenerative disease
A proportion of Amyotrophic lateral sclerosis (ALS) cases result from impaired mutant superoxide dismutase-1 (SOD1) maturation. The copper chaperone for SOD1 (hCCS) forms a transient complex with SOD1 and catalyses the final stages of its maturation. We find that a neurodegenerative disease-associat...
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| I publikationen: | Sci Rep |
|---|---|
| Huvudupphovsmän: | , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Nature Publishing Group
2016
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4901319/ https://ncbi.nlm.nih.gov/pubmed/27282955 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep27691 |
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