Brain transplants of cells expressing the carboxyl-terminal fragment of the Alzheimer amyloid protein precursor cause specific neuropathology in vivo.

Liknande verk

• Deposition of beta/A4 immunoreactivity and neuronal pathology in transgenic mice expressing the carboxyl-terminal fragment of the Alzheimer amyloid precursor in the brain.
  av: Kammesheidt, A, et al.
  Publicerad: (1992)
• Major Carboxyl terminal fragments generated by Gamma-secretase processing of the Alzheimer’s amyloid precursor are 50 and 51 aa in length
  av: Pinnix, Inga, et al.
  Publicerad: (2013)
• Extracellular vesicles: where the amyloid precursor protein carboxyl‐terminal fragments accumulate and amyloid‐β oligomerizes
  av: Pérez‐González, Rocío, et al.
  Publicerad: (2020)
• Carboxyl-terminal fragments of beta-amyloid precursor protein bind to microtubules and the associated protein tau.
  av: Islam, K., et al.
  Publicerad: (1997)
• Lysosomal dysfunction in the brain of a mouse model with intraneuronal accumulation of carboxyl terminal fragments of the amyloid precursor protein
  av: Kaur, Gurjinder, et al.
  Publicerad: (2016)