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The Drosophila HNF4 nuclear receptor promotes glucose-stimulated insulin secretion and mitochondrial function in adults

Although mutations in HNF4A were identified as the cause of Maturity Onset Diabetes of the Young 1 (MODY1) two decades ago, the mechanisms by which this nuclear receptor regulates glucose homeostasis remain unclear. Here we report that loss of Drosophila HNF4 recapitulates hallmark symptoms of MODY1...

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Bibliografske podrobnosti
izdano v:eLife
Main Authors: Barry, William E, Thummel, Carl S
Format: Artigo
Jezik:Inglês
Izdano: eLife Sciences Publications, Ltd 2016
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4869932/
https://ncbi.nlm.nih.gov/pubmed/27185732
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.11183
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