Prostaglandin E(2) constrains systemic inflammation through an innate lymphoid cell–IL-22 axis**

Systemic inflammation, resulting from massive release of pro-inflammatory molecules into the circulatory system, is a major risk factor for severe illness, but the precise mechanisms underlying its control are incompletely understood. We observed that prostaglandin E(2) (PGE(2)) through its receptor...

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Detaylı Bibliyografya
Yayımlandı:Science
Asıl Yazarlar: Duffin, Rodger, O’Connor, Richard A., Crittenden, Siobhan, Forster, Thorsten, Yu, Cunjing, Zheng, Xiaozhong, Smyth, Danielle, Robb, Calum T., Rossi, Fiona, Skouras, Christos, Tang, Shaohui, Richards, James, Pellicoro, Antonella, Weller, Richard B., Breyer, Richard M., Mole, Damian J., Iredale, John P., Anderton, Stephen M., Narumiya, Shuh, Maizels, Rick M., Ghazal, Peter, Howie, Sarah E., Rossi, Adriano G., Yao, Chengcan
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2016
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4841390/
https://ncbi.nlm.nih.gov/pubmed/26989254
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/science.aad9903
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