Prostaglandin E(2) constrains systemic inflammation through an innate lymphoid cell–IL-22 axis**

Systemic inflammation, resulting from massive release of pro-inflammatory molecules into the circulatory system, is a major risk factor for severe illness, but the precise mechanisms underlying its control are incompletely understood. We observed that prostaglandin E(2) (PGE(2)) through its receptor...

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Publicado no:Science
Main Authors: Duffin, Rodger, O’Connor, Richard A., Crittenden, Siobhan, Forster, Thorsten, Yu, Cunjing, Zheng, Xiaozhong, Smyth, Danielle, Robb, Calum T., Rossi, Fiona, Skouras, Christos, Tang, Shaohui, Richards, James, Pellicoro, Antonella, Weller, Richard B., Breyer, Richard M., Mole, Damian J., Iredale, John P., Anderton, Stephen M., Narumiya, Shuh, Maizels, Rick M., Ghazal, Peter, Howie, Sarah E., Rossi, Adriano G., Yao, Chengcan
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4841390/
https://ncbi.nlm.nih.gov/pubmed/26989254
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/science.aad9903
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