Hydrophobic motif site-phosphorylated protein kinase CβII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy

Antzeko izenburuak

• High Glucose Forces a Positive Feedback Loop Connecting Akt Kinase and FoxO1 Transcription Factor to Activate mTORC1 Kinase for Mesangial Cell Hypertrophy and Matrix Protein Expression
  nork: Das, Falguni, et al.
  Argitaratua: (2014)
• TGFβ acts through PDGFRβ to activate mTORC1 via the Akt/PRAS40 axis and causes glomerular mesangial cell hypertrophy and matrix protein expression
  nork: Maity, Soumya, et al.
  Argitaratua: (2020)
• TGFβ-Stimulated MicroRNA-21 Utilizes PTEN to Orchestrate AKT/mTORC1 Signaling for Mesangial Cell Hypertrophy and Matrix Expansion
  nork: Dey, Nirmalya, et al.
  Argitaratua: (2012)
• High glucose enhances microRNA-26a to activate mTORC1 for mesangial cell hypertrophy and matrix protein expression
  nork: Dey, Nirmalya, et al.
  Argitaratua: (2015)
• Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion
  nork: Bera, Amit, et al.
  Argitaratua: (2017)