Abrogation of Nrf2 impairs antioxidant signaling and promotes atrial hypertrophy in response to high-intensity exercise stress

BACKGROUND: Anomalies in myocardial structure involving myocyte growth, hypertrophy, differentiation, apoptosis, necrosis etc. affects its function and render cardiac tissue more vulnerable to the development of heart failure. Although oxidative stress has a well-established role in cardiac remodeli...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
הוצא לאור ב:J Transl Med
Main Authors: Kumar, Radhakrishnan Rajesh, Narasimhan, Madhusudhanan, Shanmugam, Gobinath, Hong, Jennifer, Devarajan, Asokan, Palaniappan, Sethu, Zhang, Jianhua, Halade, Ganesh V., Darley-Usmar, Victor M., Hoidal, John R., Rajasekaran, Namakkal S.
פורמט: Artigo
שפה:Inglês
יצא לאור: BioMed Central 2016
נושאים:
Research
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC4822244/
https://ncbi.nlm.nih.gov/pubmed/27048381
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12967-016-0839-3
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