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Glioblastomas with copy number gains in EGFR and RNF139 show increased expressions of carbonic anhydrase genes transformed by ENO1

BACKGROUND: Prominence of glycolysis in glioblastomas may be non-specific or a feature of oncogene-related subgroups (i.e. amplified EGFR, etc.). Relationships between amplified oncogenes and expressions of metabolic genes associated with glycolysis, directly or indirectly via pH, were therefore inv...

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Bibliografiset tiedot
Julkaisussa:BBA Clin
Päätekijät: Beckner, Marie E., Pollack, Ian F., Nordberg, Mary L., Hamilton, Ronald L.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Elsevier 2015
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4802406/
https://ncbi.nlm.nih.gov/pubmed/27051584
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbacli.2015.11.001
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