טוען...
Deletion of P2X7 attenuates hyperoxia-induced acute lung injury via inflammasome suppression
Increasing evidence shows that hyperoxia is a serious complication of oxygen therapy in acutely ill patients that causes excessive production of free radicals leading to hyperoxia-induced acute lung injury (HALI). Our previous studies have shown that P2X7 receptor activation is required for inflamma...
שמור ב:
| הוצא לאור ב: | Am J Physiol Lung Cell Mol Physiol |
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| Main Authors: | , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Physiological Society
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4796258/ https://ncbi.nlm.nih.gov/pubmed/26747786 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00417.2015 |
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