Akap1 genetic deletion increases the severity of hyperoxia-induced acute lung injury in mice

Critically ill patients are commonly treated with high levels of oxygen, hyperoxia, for prolonged periods of time. Unfortunately, extended exposure to hyperoxia can exacerbate respiratory failure and lead to a high mortality rate. Mitochondrial A-kinase anchoring protein (Akap) has been shown to reg...

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Publicado en:Am J Physiol Lung Cell Mol Physiol
Autores principales: Narala, Venkata Ramireddy, Fukumoto, Jutaro, Hernández-Cuervo, Helena, Patil, Sahebgowda Sidramagowda, Krishnamurthy, Sudarshan, Breitzig, Mason, Galam, Lakshmi, Soundararajan, Ramani, Lockey, Richard F., Kolliputi, Narasaiah
Formato: Artigo
Lenguaje:Inglês
Publicado: American Physiological Society 2018
Materias:
Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6008128/
https://ncbi.nlm.nih.gov/pubmed/29388469
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00365.2017
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