Akap1 genetic deletion increases the severity of hyperoxia-induced acute lung injury in mice

Critically ill patients are commonly treated with high levels of oxygen, hyperoxia, for prolonged periods of time. Unfortunately, extended exposure to hyperoxia can exacerbate respiratory failure and lead to a high mortality rate. Mitochondrial A-kinase anchoring protein (Akap) has been shown to reg...

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Detalhes bibliográficos
Publicado no:Am J Physiol Lung Cell Mol Physiol
Main Authors: Narala, Venkata Ramireddy, Fukumoto, Jutaro, Hernández-Cuervo, Helena, Patil, Sahebgowda Sidramagowda, Krishnamurthy, Sudarshan, Breitzig, Mason, Galam, Lakshmi, Soundararajan, Ramani, Lockey, Richard F., Kolliputi, Narasaiah
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2018
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6008128/
https://ncbi.nlm.nih.gov/pubmed/29388469
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00365.2017
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