Akap1 genetic deletion increases the severity of hyperoxia-induced acute lung injury in mice

Critically ill patients are commonly treated with high levels of oxygen, hyperoxia, for prolonged periods of time. Unfortunately, extended exposure to hyperoxia can exacerbate respiratory failure and lead to a high mortality rate. Mitochondrial A-kinase anchoring protein (Akap) has been shown to reg...

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Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Am J Physiol Lung Cell Mol Physiol
Κύριοι συγγραφείς: Narala, Venkata Ramireddy, Fukumoto, Jutaro, Hernández-Cuervo, Helena, Patil, Sahebgowda Sidramagowda, Krishnamurthy, Sudarshan, Breitzig, Mason, Galam, Lakshmi, Soundararajan, Ramani, Lockey, Richard F., Kolliputi, Narasaiah
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Physiological Society 2018
Θέματα:
Research Article
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6008128/
https://ncbi.nlm.nih.gov/pubmed/29388469
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00365.2017
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