Akap1 genetic deletion increases the severity of hyperoxia-induced acute lung injury in mice

Critically ill patients are commonly treated with high levels of oxygen, hyperoxia, for prolonged periods of time. Unfortunately, extended exposure to hyperoxia can exacerbate respiratory failure and lead to a high mortality rate. Mitochondrial A-kinase anchoring protein (Akap) has been shown to reg...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
הוצא לאור ב:Am J Physiol Lung Cell Mol Physiol
Main Authors: Narala, Venkata Ramireddy, Fukumoto, Jutaro, Hernández-Cuervo, Helena, Patil, Sahebgowda Sidramagowda, Krishnamurthy, Sudarshan, Breitzig, Mason, Galam, Lakshmi, Soundararajan, Ramani, Lockey, Richard F., Kolliputi, Narasaiah
פורמט: Artigo
שפה:Inglês
יצא לאור: American Physiological Society 2018
נושאים:
Research Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC6008128/
https://ncbi.nlm.nih.gov/pubmed/29388469
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00365.2017
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