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Does inactivation of USP14 enhance degradation of proteasomal substrates that are associated with neurodegenerative diseases?
A common pathological hallmark of age-related neurodegenerative diseases is the intracellular accumulation of protein aggregates such as α-synuclein in Parkinson’s disease, TDP-43 in ALS, and tau in Alzheimer’s disease. Enhancing intracellular clearance of aggregation-prone proteins is a plausible s...
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| Publicado no: | F1000Res |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
F1000Research
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4792207/ https://ncbi.nlm.nih.gov/pubmed/26998235 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12688/f1000research.7800.2 |
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