High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model

There is growing evidence of the involvement of advanced glycation end products (AGEs) in the pathogenesis of neurodegenerative processes including Alzheimer's disease (AD) and their function as a seed for the aggregation of Aβ, a hallmark feature of AD. AGEs are formed endogenously and exogeno...

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Publicat a:Aging Cell
Autors principals: Lubitz, Irit, Ricny, Jan, Atrakchi‐Baranes, Dana, Shemesh, Chen, Kravitz, Efrat, Liraz‐Zaltsman, Sigal, Maksin‐Matveev, Anna, Cooper, Itzik, Leibowitz, Avshalom, Uribarri, Jaime, Schmeidler, James, Cai, Weijing, Kristofikova, Zdena, Ripova, Daniela, LeRoith, Derek, Schnaider‐Beeri, Michal
Format: Artigo
Idioma:Inglês
Publicat: John Wiley and Sons Inc. 2016
Matèries:
Original Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783343/
https://ncbi.nlm.nih.gov/pubmed/26781037
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12436
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