High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model

There is growing evidence of the involvement of advanced glycation end products (AGEs) in the pathogenesis of neurodegenerative processes including Alzheimer's disease (AD) and their function as a seed for the aggregation of Aβ, a hallmark feature of AD. AGEs are formed endogenously and exogeno...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Aging Cell
Κύριοι συγγραφείς: Lubitz, Irit, Ricny, Jan, Atrakchi‐Baranes, Dana, Shemesh, Chen, Kravitz, Efrat, Liraz‐Zaltsman, Sigal, Maksin‐Matveev, Anna, Cooper, Itzik, Leibowitz, Avshalom, Uribarri, Jaime, Schmeidler, James, Cai, Weijing, Kristofikova, Zdena, Ripova, Daniela, LeRoith, Derek, Schnaider‐Beeri, Michal
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: John Wiley and Sons Inc. 2016
Θέματα:
Original Articles
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783343/
https://ncbi.nlm.nih.gov/pubmed/26781037
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12436
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