High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model

There is growing evidence of the involvement of advanced glycation end products (AGEs) in the pathogenesis of neurodegenerative processes including Alzheimer's disease (AD) and their function as a seed for the aggregation of Aβ, a hallmark feature of AD. AGEs are formed endogenously and exogeno...

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محفوظ في:
التفاصيل البيبلوغرافية
الحاوية / القاعدة:Aging Cell
المؤلفون الرئيسيون: Lubitz, Irit, Ricny, Jan, Atrakchi‐Baranes, Dana, Shemesh, Chen, Kravitz, Efrat, Liraz‐Zaltsman, Sigal, Maksin‐Matveev, Anna, Cooper, Itzik, Leibowitz, Avshalom, Uribarri, Jaime, Schmeidler, James, Cai, Weijing, Kristofikova, Zdena, Ripova, Daniela, LeRoith, Derek, Schnaider‐Beeri, Michal
التنسيق: Artigo
اللغة:Inglês
منشور في: John Wiley and Sons Inc. 2016
الموضوعات:
Original Articles
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783343/
https://ncbi.nlm.nih.gov/pubmed/26781037
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12436
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