Intracranial delivery of Interleukin-17A via adeno-associated virus fails to induce physical and learning disabilities and neuroinflammation in mice but improves glucose metabolism through AKT signaling pathway

Interleukin-17A (IL-17A) is generally considered as one of the pathogenic factors involved in multiple sclerosis (MS). Indirect evidence for this is that IL-17A-producing T helper 17 (Th17) cells preferentially accumulate in lesions of MS and experimental autoimmune encephalomyelitis (EAE). However,...

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I publikationen:Brain Behav Immun
Huvudupphovsmän: Yang, Junling, Kou, Jinghong, Lim, Jeong-Eun, Lalonde, Robert, Fukuchi, Ken-ichiro
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2015
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783216/
https://ncbi.nlm.nih.gov/pubmed/26562537
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbi.2015.11.005
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