IGF-1 Prevents Diastolic and Systolic Dysfunction Associated with Cardiomyopathy and Preserves Adrenergic Sensitivity

Podobne zapisy

• Nitric Oxide-Dependent Activation of CaMKII Increases Diastolic Sarcoplasmic Reticulum Calcium Release in Cardiac Myocytes in Response to Adrenergic Stimulation
  od: Curran, Jerry, i wsp.
  Wydane: (2014)
• Effects of increased systolic Ca(2+) and phospholamban phosphorylation during β-adrenergic stimulation on Ca(2+) transient kinetics in cardiac myocytes
  od: Roof, Steve R., i wsp.
  Wydane: (2011)
• Effects of increased systolic Ca(2+) and β-adrenergic stimulation on Ca(2+) transient decline in NOS1 knockout cardiac myocytes
  od: Roof, Steve R, i wsp.
  Wydane: (2012)
• Systolic-diastolic functional coupling in healthy children and in those with dilated cardiomyopathy
  od: Friedberg, Mark K., i wsp.
  Wydane: (2016)
• Diastolic dysfunction in hypertrophic cardiomyopathy. Effect on active force generation during systole.
  od: Gwathmey, J K, i wsp.
  Wydane: (1991)