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PARP1 Links CHD2-Mediated Chromatin Expansion and H3.3 Deposition to DNA Repair by Non-homologous End-Joining

The response to DNA double-strand breaks (DSBs) requires alterations in chromatin structure to promote the assembly of repair complexes on broken chromosomes. Non-homologous end-joining (NHEJ) is the dominant DSB repair pathway in human cells, but our understanding of how it operates in chromatin is...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Mol Cell
Päätekijät: Luijsterburg, Martijn S., de Krijger, Inge, Wiegant, Wouter W., Shah, Rashmi G., Smeenk, Godelieve, de Groot, Anton J.L., Pines, Alex, Vertegaal, Alfred C.O., Jacobs, Jacqueline J.L., Shah, Girish M., van Attikum, Haico
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Cell Press 2016
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4769320/
https://ncbi.nlm.nih.gov/pubmed/26895424
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2016.01.019
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