PKCδ inhibition normalizes the wound-healing capacity of diabetic human fibroblasts

Abnormal fibroblast function underlies poor wound healing in patients with diabetes; however, the mechanisms that impair wound healing are poorly defined. Here, we evaluated fibroblasts from individuals who had type 1 diabetes (T1D) for 50 years or more (Medalists, n = 26) and from age-matched contr...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:J Clin Invest
Κύριοι συγγραφείς: Khamaisi, Mogher, Katagiri, Sayaka, Keenan, Hillary, Park, Kyoungmin, Maeda, Yasutaka, Li, Qian, Qi, Weier, Thomou, Thomas, Eschuk, Danielle, Tellechea, Ana, Veves, Aris, Huang, Chenyu, Orgill, Dennis Paul, Wagers, Amy, King, George L.
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Society for Clinical Investigation 2016
Θέματα:
Research Article
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4767341/
https://ncbi.nlm.nih.gov/pubmed/26808499
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI82788
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