Altered epidermal lipid processing and calcium distribution in the KID syndrome mouse model Cx26S17F

The keratitis–ichthyosis–deafness (KID) syndrome is caused by mutations in the gap junctional channel protein connexin 26 (Cx26), among them the mutation Cx26S17F. Heterozygous Cx26S17F mice resemble the human KID syndrome, i.e. exhibiting epidermal hyperplasia and hearing impairments. Newborn Cx26S...

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Publicat a:FEBS Lett
Autors principals: Bosen, Felicitas, Celli, Anna, Crumrine, Debra, vom Dorp, Katharina, Ebel, Philipp, Jastrow, Holger, Dörmann, Peter, Winterhager, Elke, Mauro, Theodora, Willecke, Klaus
Format: Artigo
Idioma:Inglês
Publicat: 2015
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4741282/
https://ncbi.nlm.nih.gov/pubmed/26070424
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.febslet.2015.05.047
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