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A genetic variant of cortactin linked to acute lung injury impairs lamellipodia dynamics and endothelial wound healing
Inflammatory mediators released in acute lung injury (ALI) trigger the disruption of interendothelial junctions, leading to loss of vascular barrier function, protein-rich pulmonary edema, and severe hypoxemia. Genetic signatures that predict patient recovery or disease progression are poorly define...
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| 出版年: | Am J Physiol Lung Cell Mol Physiol |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4628987/ https://ncbi.nlm.nih.gov/pubmed/26361873 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00062.2015 |
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