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Targeting mitochondrial reactive oxygen species to modulate hypoxia-induced pulmonary hypertension
OBJECTIVE: Pulmonary hypertension (PH) is characterized by increased pulmonary vascular remodeling, resistance, and pressures. Reactive oxygen species (ROS) contribute to PH-associated vascular dysfunction. NADPH oxidases (Nox) and mitochondria are major sources of superoxide (O(2)(•−)) and hydrogen...
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| Publié dans: | Free Radic Biol Med |
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| Auteurs principaux: | , , , , , , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2015
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4615392/ https://ncbi.nlm.nih.gov/pubmed/26073127 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2015.05.042 |
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