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Prion Protein Prolines 102 and 105 and the Surrounding Lysine Cluster Impede Amyloid Formation
Human prion diseases can have acquired, sporadic, or genetic origins, each of which results in the conversion of prion protein (PrP) to transmissible, pathological forms. The genetic prion disease Gerstmann-Straussler-Scheinker syndrome can arise from point mutations of prolines 102 or 105. However,...
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| Publicado no: | J Biol Chem |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4571877/ https://ncbi.nlm.nih.gov/pubmed/26175152 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M115.665844 |
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