Genetic deficit of K(Ca)3.1 channels protects against pulmonary circulatory collapse induced by TRPV4 channel activation

BACKGROUND AND PURPOSE: The intermediate conductance calcium/calmodulin-regulated K(+) channel K(Ca)3.1 produces hyperpolarizing K(+) currents that counteract depolarizing currents carried by transient receptor potential (TRP) channels, and provide the electrochemical driving force for Cl(−) and flu...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Br J Pharmacol
Päätekijät: Wandall-Frostholm, Christine, Dalsgaard, Thomas, Bajoriūnas, Vytis, Oliván-Viguera, Aida, Sadda, Veeruanjaneyulu, Beck, Lilliana, Mogensen, Susie, Stankevicius, Edgaras, Simonsen, Ulf, Köhler, Ralf
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: John Wiley & Sons, Ltd 2015
Aiheet:
Research Papers
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4562510/
https://ncbi.nlm.nih.gov/pubmed/26102209
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/bph.13234
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