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Genetic deficit of K(Ca)3.1 channels protects against pulmonary circulatory collapse induced by TRPV4 channel activation
BACKGROUND AND PURPOSE: The intermediate conductance calcium/calmodulin-regulated K(+) channel K(Ca)3.1 produces hyperpolarizing K(+) currents that counteract depolarizing currents carried by transient receptor potential (TRP) channels, and provide the electrochemical driving force for Cl(−) and flu...
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| 出版年: | Br J Pharmacol |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley & Sons, Ltd
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4562510/ https://ncbi.nlm.nih.gov/pubmed/26102209 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/bph.13234 |
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