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Additive neuroprotection of a 20-HETE inhibitor with delayed therapeutic hypothermia after hypoxia-ischemia in neonatal piglets
The severity of perinatal hypoxia-ischemia and the delay in initiating therapeutic hypothermia limit the efficacy of hypothermia. After hypoxia-ischemia in neonatal piglets, the arachidonic acid metabolite, 20-hydroxyeicosatetraenoic acid (20-HETE), has been found to contribute to oxidative stress a...
Tallennettuna:
| Julkaisussa: | Dev Neurosci |
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| Päätekijät: | , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2015
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4514546/ https://ncbi.nlm.nih.gov/pubmed/25721266 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000369007 |
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