טוען...
Regulation of Pro-Apoptotic Phosphorylation of Kv2.1 K(+) Channels
Caspase activity during apoptosis is inhibited by physiological concentrations of intracellular K(+). To enable apoptosis in injured cortical and hippocampal neurons, cellular loss of this cation is facilitated by the insertion of Kv2.1 K(+) channels into the plasma membrane via a Zn(2+)/CaMKII/SNAR...
שמור ב:
| הוצא לאור ב: | PLoS One |
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| Main Authors: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Public Library of Science
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4482604/ https://ncbi.nlm.nih.gov/pubmed/26115091 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0129498 |
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