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Endogenous murine Aβ increases amyloid deposition in APP23 but not in APPPS1 transgenic mice
Endogenous murine amyloid-β peptide (Aβ) is expressed in most Aβ precursor protein (APP) transgenic mouse models of Alzheimer's disease but its contribution to β-amyloidosis remains unclear. We demonstrate ~35% increased cerebral Aβ load in APP23 transgenic mice compared to age-matched APP23 mi...
שמור ב:
| הוצא לאור ב: | Neurobiol Aging |
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| Main Authors: | , , , , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4457564/ https://ncbi.nlm.nih.gov/pubmed/25911278 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neurobiolaging.2015.03.011 |
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