The Ca(2+)-activated cation channel TRPM4 is a negative regulator of angiotensin II-induced cardiac hypertrophy

Cardiac muscle adapts to hemodynamic stress by altering myocyte size and function, resulting in cardiac hypertrophy. Alteration in myocyte calcium homeostasis is known to be an initial signal in cardiac hypertrophy signaling. Transient receptor potential melastatin 4 protein (TRPM4) is a calcium-act...

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Bibliographic Details
Published in:Basic Res Cardiol
Main Authors: Kecskés, Miklós, Jacobs, Griet, Kerselaers, Sara, Syam, Ninda, Menigoz, Aurélie, Vangheluwe, Peter, Freichel, Marc, Flockerzi, Veit, Voets, Thomas, Vennekens, Rudi
Format: Artigo
Language:Inglês
Published: Springer Berlin Heidelberg 2015
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Original Contribution
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC4456993/
https://ncbi.nlm.nih.gov/pubmed/26043922
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-015-0501-x
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