The Ca(2+)-activated cation channel TRPM4 is a negative regulator of angiotensin II-induced cardiac hypertrophy

Cardiac muscle adapts to hemodynamic stress by altering myocyte size and function, resulting in cardiac hypertrophy. Alteration in myocyte calcium homeostasis is known to be an initial signal in cardiac hypertrophy signaling. Transient receptor potential melastatin 4 protein (TRPM4) is a calcium-act...

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Detaylı Bibliyografya
Yayımlandı:Basic Res Cardiol
Asıl Yazarlar: Kecskés, Miklós, Jacobs, Griet, Kerselaers, Sara, Syam, Ninda, Menigoz, Aurélie, Vangheluwe, Peter, Freichel, Marc, Flockerzi, Veit, Voets, Thomas, Vennekens, Rudi
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Springer Berlin Heidelberg 2015
Konular:
Original Contribution
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4456993/
https://ncbi.nlm.nih.gov/pubmed/26043922
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00395-015-0501-x
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