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Metabolism of Doxorubicin to the Cardiotoxic Metabolite Doxorubicinol Is Increased in a Mouse Model of Chronic Glutathione Deficiency: A Potential Role for Carbonyl Reductase 3

Doxorubicin is highly effective at inducing DNA double-strand breaks in rapidly dividing cells, which has led to it being a widely used cancer chemotherapeutic. However, clinical administration of doxorubicin is limited by off-target cardiotoxicity, which is thought to be mediated by doxorubicinol,...

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Vydáno v:Chem Biol Interact
Hlavní autoři: Schaupp, Christopher M., White, Collin C., Merrill, Gary F., Kavanagh, Terrance J.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2014
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4414687/
https://ncbi.nlm.nih.gov/pubmed/25446851
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cbi.2014.11.010
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