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Angiotensin II limits NO production by upregulating arginase through a p38 MAPK - ATF-2 pathway
Enhanced vascular arginase activity can impair endothelium-dependent vasorelaxation by decreasing L-arginine availability to endothelial nitric oxide (NO) synthase, thereby reducing NO production and uncoupling NOS function. Elevated angiotensin II (Ang II) is a key component of endothelial dysfunct...
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| Yayımlandı: | Eur J Pharmacol |
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| Asıl Yazarlar: | , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
2014
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4412038/ https://ncbi.nlm.nih.gov/pubmed/25446432 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ejphar.2014.10.042 |
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