Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway

Enhanced vascular arginase activity impairs endothelium-dependent vasorelaxation by decreasing l-arginine availability to endothelial nitric oxide (NO) synthase, thereby reducing NO production. Elevated angiotensin II (ANG II) is a key component of endothelial dysfunction in many cardiovascular dise...

ver descrição completa

Na minha lista:
Detalhes bibliográficos
Main Authors: Shatanawi, Alia, Romero, Maritza J., Iddings, Jennifer A., Chandra, Surabhi, Umapathy, Nagavedi S., Verin, Alexander D., Caldwell, Ruth B., Caldwell, R. William
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2011
Assuntos:
Vascular Biology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093945/
https://ncbi.nlm.nih.gov/pubmed/21289285
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00328.2010
Tags: Adicionar Tag
Sem tags, seja o primeiro a adicionar uma tag!
A carregar...

Registos relacionados