Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway

Enhanced vascular arginase activity impairs endothelium-dependent vasorelaxation by decreasing l-arginine availability to endothelial nitric oxide (NO) synthase, thereby reducing NO production. Elevated angiotensin II (ANG II) is a key component of endothelial dysfunction in many cardiovascular dise...

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Hlavní autoři: Shatanawi, Alia, Romero, Maritza J., Iddings, Jennifer A., Chandra, Surabhi, Umapathy, Nagavedi S., Verin, Alexander D., Caldwell, Ruth B., Caldwell, R. William
Médium: Artigo
Jazyk:Inglês
Vydáno: American Physiological Society 2011
Témata:
Vascular Biology
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093945/
https://ncbi.nlm.nih.gov/pubmed/21289285
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00328.2010
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