Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway

Enhanced vascular arginase activity impairs endothelium-dependent vasorelaxation by decreasing l-arginine availability to endothelial nitric oxide (NO) synthase, thereby reducing NO production. Elevated angiotensin II (ANG II) is a key component of endothelial dysfunction in many cardiovascular dise...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Shatanawi, Alia, Romero, Maritza J., Iddings, Jennifer A., Chandra, Surabhi, Umapathy, Nagavedi S., Verin, Alexander D., Caldwell, Ruth B., Caldwell, R. William
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Physiological Society 2011
Gaiak:
Vascular Biology
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093945/
https://ncbi.nlm.nih.gov/pubmed/21289285
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00328.2010
Etiketak: Etiketa erantsi
Etiketarik gabe, Izan zaitez lehena erregistro honi etiketa jartzen!

Antzeko izenburuak