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Impaired calcium-calmodulin-dependent inactivation of Ca(v)1.2 contributes to loss of sarcoplasmic reticulum calcium release refractoriness in mice lacking calsequestrin 2
AIMS: In cardiac muscle, Ca(2+) release from sarcoplasmic reticulum (SR) is reduced with successively shorter coupling intervals of premature stimuli, a phenomenon known as SR Ca(2+) release refractoriness. We recently reported that the SR luminal Ca(2+) binding protein calsequestrin 2 (Casq2) contr...
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| 發表在: | J Mol Cell Cardiol |
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| Main Authors: | , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
2015
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4405490/ https://ncbi.nlm.nih.gov/pubmed/25758429 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2015.02.027 |
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