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Impaired calcium-calmodulin-dependent inactivation of Ca(v)1.2 contributes to loss of sarcoplasmic reticulum calcium release refractoriness in mice lacking calsequestrin 2

AIMS: In cardiac muscle, Ca(2+) release from sarcoplasmic reticulum (SR) is reduced with successively shorter coupling intervals of premature stimuli, a phenomenon known as SR Ca(2+) release refractoriness. We recently reported that the SR luminal Ca(2+) binding protein calsequestrin 2 (Casq2) contr...

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Bibliografske podrobnosti
izdano v:J Mol Cell Cardiol
Main Authors: Kryshtal, Dmytro O., Gryshchenko, Oleksiy, Gomez-Hurtado, Nieves, Knollmann, Bjorn C.
Format: Artigo
Jezik:Inglês
Izdano: 2015
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4405490/
https://ncbi.nlm.nih.gov/pubmed/25758429
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2015.02.027
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