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Inhibitor-κB kinase attenuates Hsp90-dependent endothelial nitric oxide synthase function in vascular endothelial cells
Endothelial nitric oxide (NO) synthase (eNOS) is the predominant isoform that generates NO in the blood vessels. Many different regulators, including heat shock protein 90 (Hsp90), govern eNOS function. Hsp90-dependent phosphorylation of eNOS is a critical event that determines eNOS activity. In our...
Wedi'i Gadw mewn:
| Cyhoeddwyd yn: | Am J Physiol Cell Physiol |
|---|---|
| Prif Awduron: | , , , , , , , |
| Fformat: | Artigo |
| Iaith: | Inglês |
| Cyhoeddwyd: |
American Physiological Society
2015
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| Pynciau: | |
| Mynediad Ar-lein: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4398846/ https://ncbi.nlm.nih.gov/pubmed/25652452 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00367.2014 |
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