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Prolactin stimulates sodium and chloride ion channels in A6 renal epithelial cells
Many hormonal pathways contribute to the regulation of renal epithelial sodium channel (ENaC) function, a key process for maintaining blood volume and controlling blood pressure. In the present study, we examined whether the peptide hormone prolactin (PRL) regulates ENaC function in renal epithelial...
Tallennettuna:
| Julkaisussa: | Am J Physiol Renal Physiol |
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| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Physiological Society
2015
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4385885/ https://ncbi.nlm.nih.gov/pubmed/25587116 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00270.2014 |
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