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Arrestin-3-dependent activation of c-Jun N-terminal kinases (JNKs)
Only one out of four arrestin subtypes expressed in mammals, arrestin-3, facilitates the activation of JNK family kinases. Here we describe two different paradigms that allow the elucidation of the mechanisms involved. One is based on reconstitution of signaling modules from purified proteins: arres...
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| Vydáno v: | Curr Protoc Pharmacol |
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| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4361079/ https://ncbi.nlm.nih.gov/pubmed/25737158 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/0471141755.ph0212s68 |
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