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Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III
Excessive mitochondrial reactive oxygen species (ROS) emission is a critical component in the etiology of ischemic injury. Complex I and complex III of the electron transport chain are considered the primary sources of ROS emission during cardiac ischemia and reperfusion (IR) injury. Several factors...
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| Yayımlandı: | Front Physiol |
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| Asıl Yazarlar: | , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Frontiers Media S.A.
2015
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4354303/ https://ncbi.nlm.nih.gov/pubmed/25805998 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphys.2015.00058 |
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