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Mitochondrial handling of excess Ca(2+) is substrate-dependent with implications for reactive oxygen species generation
AIM: The mitochondrial electron transport chain is the major source of reactive oxygen species (ROS) during cardiac ischemia. Several mechanisms modulate ROS production; one is mitochondrial Ca(2+) uptake. Here we sought to elucidate the effects of extra-mitochondrial Ca(2+) (e[Ca(2+)]) on ROS produ...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3542420/ https://ncbi.nlm.nih.gov/pubmed/23010495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2012.09.020 |
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