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Defective PDI release from platelets and endothelial cells impairs thrombus formation in Hermansky-Pudlak syndrome
Protein disulfide isomerase (PDI), secreted from platelets and endothelial cells after injury, is required for thrombus formation. The effect of platelet and endothelial cell granule contents on PDI-mediated thrombus formation was studied by intravital microscopy using a mouse model of Hermansky-Pud...
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| Vydáno v: | Blood |
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| Hlavní autoři: | , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society of Hematology
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4351508/ https://ncbi.nlm.nih.gov/pubmed/25593336 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2014-08-597419 |
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