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IER3 supports KRAS(G12D)-dependent pancreatic cancer development by sustaining ERK1/2 phosphorylation
Activating mutations in the KRAS oncogene are prevalent in pancreatic ductal adenocarcinoma (PDAC). We previously demonstrated that pancreatic intraepithelial neoplasia (PanIN) formation, which precedes malignant transformation, associates with the expression of immediate early response 3 (Ier3) as...
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| 出版年: | J Clin Invest |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Clinical Investigation
2014
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4347237/ https://ncbi.nlm.nih.gov/pubmed/25250570 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI76037 |
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