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The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses

Telomeres, the ends of our linear chromosomes, can function as ‘replicometers’, capable of counting cell division cycles as they progressively erode with every round of DNA replication. Once they are critically short, telomeres become dysfunctional and consequently activate a proliferative arrest ca...

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Bibliografske podrobnosti
izdano v:Aging Cell
Main Authors: Suram, Anitha, Herbig, Utz
Format: Artigo
Jezik:Inglês
Izdano: BlackWell Publishing Ltd 2014
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4331758/
https://ncbi.nlm.nih.gov/pubmed/25040628
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12246
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