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The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses
Telomeres, the ends of our linear chromosomes, can function as ‘replicometers’, capable of counting cell division cycles as they progressively erode with every round of DNA replication. Once they are critically short, telomeres become dysfunctional and consequently activate a proliferative arrest ca...
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| Publicado no: | Aging Cell |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BlackWell Publishing Ltd
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4331758/ https://ncbi.nlm.nih.gov/pubmed/25040628 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12246 |
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