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Epithelial sodium channel abundance is decreased by an unfolded protein response induced by hyperosmolality
Large shifts of osmolality occur in the kidney medulla as part of the urine concentrating mechanism. Hyperosmotic stress profoundly challenges cellular homeostasis and induces endoplasmic reticulum (ER) stress. Here, we examined the unfolded protein response (UPR) in hyperosmotically‐challenged prin...
Tallennettuna:
| Julkaisussa: | Physiol Rep |
|---|---|
| Päätekijät: | , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Wiley Periodicals, Inc.
2014
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4255800/ https://ncbi.nlm.nih.gov/pubmed/25413317 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.12169 |
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