ATMIN is required for the ATM-mediated signaling and recruitment of 53BP1 to DNA damage sites upon replication stress

Unresolved replication intermediates can block the progression of replication forks and become converted into DNA lesions, hence exacerbating genomic instability. The p53-binding protein 1 (53BP1) forms nuclear bodies at sites of unrepaired DNA lesions to shield these regions against erosion, in a m...

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Bibliografische gegevens
Gepubliceerd in:DNA Repair (Amst)
Hoofdauteurs: Schmidt, Luisa, Wiedner, Marc, Velimezi, Georgia, Prochazkova, Jana, Owusu, Michel, Bauer, Sabine, Loizou, Joanna I.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Elsevier 2014
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Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4251980/
https://ncbi.nlm.nih.gov/pubmed/25262557
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.dnarep.2014.09.001
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